For Mother’s Day, we posted a study about maternal-fetal stress causing telomere attrition
Now, here’s an article linking stress to telomere damage in older men:
And we have similar studies on children, adolescents, and mothers of disabled kids. In fact, many years and many millions of dollars will be invested in the next decades to prove the obvious: that all cells need to copy, that telomere DNA shortens when they do, that “stress” and a bunch of other things can adversely affect their repair… and yet somehow they always conclude that further study is needed.
Currently, 16,000 studies linking telomere dysfunction to nearly every disease that exists. Why do we need these? Read my ridiculously simple theory about aging. I believe there may be only one disease and that it is worth studying therapies that actually might DO something to slow or reverse telomere damage, like TA-65 for example. See ProBonoTrials.org for more info (under construction).
This endless, incremental, reiterative, and staid method of advancing knowledge is challenged by what I call the “Ginsu Principle”. You could sell many distinct knives for cutting bread, aluminum cans, tomatoes and wood. And you can have different theories for how these knives cut those substances. Or you could just accept that a knife can cut a lot of things because it’s sharp. Our research into telomere biology violates the Ginsu Principle in a big way.
Of course, the original Ginsu Principle was called Occam’s Razor and it states that if one explanation will suffice, don’t evoke many different ones.