
What if aging was meant to kill women?
Dying young but with perfect babies is sustainable and advantaged; dying old but with abnormal babies is an evolutionary dead end.

Dying young but with perfect babies is sustainable and advantaged; dying old but with abnormal babies is an evolutionary dead end.
I was at an ideas festival filled with some bright and accomplished people and I found myself rebuked by what I would consider true ignorance at least three times.

Liz Parrish, founder of BioViva Sciences discusses the objective improvements in her biomarkers after taking gene therapy for telomerase and follastatin.

In my search for wisdom about aging, I maintain that the primary cause is telomere shortening. This does not mean that methylation, signaling with stem cell produced vesicles (like packages with a purpose) don’t play a secondary role.
I would like to recommend the TV show, Younger for its new take on telling women’s stories. Let’s hear it for the women who bravely face the challenges of balancing reproductive capacity, career, freedom, and having to tolerate men.

I have noted that over the last nine years, taking telomerase activators in my patients can lead to increases but that this does not result in continuous lengthening. Now, we understand why this is the case.