The inadequate lengthening of telomeres by telomerase allows for mutations to occur. But after carcinogenesis has occurred, the ongoing action of telomerase in those immortal cells allows for lengthening, thereby obscuring the relationship between median telomere length and cancer risk.
For #WebinarWednesday, I want to post a 16-minute interview I did recently with author and thought-leader, G. Edward Griffin. In this though-provoking exchange, I explain where humanity is going and why we can live hundreds of years.
I predict that no one is every going to collect Social Security benefits although we will probably keep paying into the system. (Sorry Ben Franklin, it’s really only taxes we can count on now)
My arthritis has gone by 90%, I am more positive, look healthier, have a better appetite and digestion, more stamina, LPa has dropped 500 points, B12 is normal , iron levels normal, Bilirubin now normal. After 40 years of abnormal readings.
-Anne N. (New Zealand)
For today’s #PhilanthropyFriday, let’s talk about what makes us centered, grounded, and flowing. It is the union of thought and breath in a properly aligned body, free of constricted flow.
Was George Washington the first or the ninth president of the United States? Despite considering myself a relatively informed person, I wasn’t even AWARE there was a debate, let alone a “conspiracy theory” tag associated with this question.
Simply stated, telomerase is neither pro nor anti-tumorigenic any more than a foot or a brake pedal cause car accidents while driving down a mountain. Telomerase, like a driver’s foot, prevents carcinogenesis by speeding up or slowing down the car.
